By B. Greg Brown, Xue Qiao Zhao, Drew Poulin, John J. Albers (auth.), Linda L. Gallo (eds.)
The Fourteenth Washington foreign Spring Symposium, held in Washington, D.C., in June 1994, introduced jointly over four hundred major scientists from 21 international locations to check and replace study on heart problems. This workforce chuffed the symposium objectives of formulating a extra finished and built-in photo of the occasions contributing to atherosclerosis and of exploring transformed gene expression as an to realizing the factors of atherosclerosis and offering clues to the technique prevention and therapy. This quantity comprises many of the papers offered on the 8 plenary periods including chosen contributions from the exact periods. The multidisciplinary nature of the chapters and their authors should still stimulate the pursuits of biochemists, mobilephone and molecular biologists, pathologists, pharmacologists, epidemiologists, nutritionists, and clinicians. the amount is split into 8 sections which mirror the point of interest of the plenary periods. half I specializes in the pathophysiology of atherosclerotic plaques and predicts that the character of the fibrous cap of atheroma determines plaque disruption and scientific occasions. Papers partially II care for atherogenic lipoproteins. The introductory paper reports the present view of the position of plasma lipoproteins in atherosclerosis. With recognize to the more recent individuals at the list,: [oxidized LDL, Lp(a)] facts is supplied that means the involvement of 1 significant gene within the improvement of oxidized LDL lipids. the expression of inflammatory genes, and the advance of aortic fatty streaks.
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Additional resources for Cardiovascular Disease: Cellular and Molecular Mechanisms, Prevention, and Treatment
Several methods including electrophoresis, hydrated density, gradient gel electrophoresis, and affinity chromatography have been employed to separate and characterize the lipoprotein particles within HDL (40,42,43). The most effective current method available to classify lipoprotein particles in HDL is based on apolipoprotein composition (39). The two major apoA-I containing lipoprotein particles within HDL classified by apolipoprotein composition are lipoprotein containing only apoA-I or both apoA-I and apoA-II, designated LpA-I and LpA-I:A-1I respectively.
Cushing and AM. Fogelman, Monocytes may amplify their recruitment into inflammatory lesions by inducing monocyte chemotactic protein (MCP-l), Arteriosclerosis and Thrombosis 12:78 (1992). 10. W. L. Svenson, M. I. M. A. Edwards, Mechanisms controlling competence gene expression in murine fibroblasts stimulated with minimally modified low density lipoprotein, Arteriosclerosis and Thrombosis 12:800 (1992). 11. M. Y. I. C. Drinkwater, H. Laks, and AM. Fogelman, A new antiinflammatory compound, leumedin, inhibits modification of low density lipoprotein, and the resulting monocyte transmigration into the subendothelial space of co-cultures of human aortic wall cells, J Clin Invest 91:1225 (1993).
Fogelman, Minimally modified low density lipoprotein is biologically active in vivo in mice, J CUn Invest 87:2253 (1991). 9. D. Cushing and AM. Fogelman, Monocytes may amplify their recruitment into inflammatory lesions by inducing monocyte chemotactic protein (MCP-l), Arteriosclerosis and Thrombosis 12:78 (1992). 10. W. L. Svenson, M. I. M. A. Edwards, Mechanisms controlling competence gene expression in murine fibroblasts stimulated with minimally modified low density lipoprotein, Arteriosclerosis and Thrombosis 12:800 (1992).